Which is a postural deviation/contracture listed for High Lumbar L1-L2?

• A. Knee flexion and ankle plantarflexion contractures
• B. Hip flexion contractures
• C. Increased lumbar lordosis
• D. Club foot

Answer: (A)

When a lesion is at the high lumbar level (L1–L2), the muscles below that level that normally extend the knee and dorsiflex the ankle are the ones most affected. The knee extensor muscles (the quadriceps) rely on nerves entering around L2–L4, so a high lumbar lesion weakens or abolishes knee extension. Without a firm knee extension, the knee tends to remain in a flexed position, creating a knee flexion contracture. At the same time, the muscles that lift the foot (the dorsiflexors) are compromised, so the unopposed plantarflexors pull the foot downward into plantarflexion, leading to an ankle plantarflexion contracture. Together, these two patterns—knee flexion and ankle plantarflexion contractures—fit the typical posture seen with high lumbar L1–L2 lesions.

Hip flexion contractures would imply persistent tightness or overactivity of the hip flexors, which is not the hallmark pattern for this level. Club foot is a broader foot deformity that can occur with various levels and conditions, but it is not the characteristic contracture listed for L1–L2 specifically. Increased lumbar lordosis is a general postural change that can occur with several spinal levels and is not the distinctive contracture combination described here.